A Practical, Evidence-Tiered Guide to What Works, What Is Promising, and What to Skip
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If you have ever stood in a supplement aisle and stared at the wall of bottles promising to "fight inflammation," you already know the problem. Almost every label is making the same claim, and almost no two ingredients have the same evidence behind them. Most ranking articles flatten this. They list ten or fifteen supplements side by side and treat fish oil and spirulina as if they belong in the same conversation, which makes it impossible to figure out where to actually start.
This guide takes a different approach. Instead of cataloging supplements by popularity or by mechanism, you will see them organized by the quality of the human clinical evidence behind each one. The supplements at the top of this list have multiple randomized controlled trials and meta-analyses showing they can move inflammatory biomarkers in real people. The ones lower down are interesting but less proven. And a few of the supplements you might expect to see here did not make the list at all, for reasons we will get into.
A quick framing note before you dig in. Chronic, low-grade inflammation is associated with a long list of conditions that show up later in life — cardiovascular disease, type 2 diabetes, and several cancers among them — and the National Cancer Institute describes how persistent inflammatory signaling can contribute to DNA damage and disease progression. That is why so many people are looking for ways to lower it. Supplements can play a role, but they work best alongside the basics: sleep, regular movement, a diet built around whole foods, and stress management. None of these supplements is a shortcut, and none of them replaces medical care for a diagnosed condition.
One more thing before you read on. This article is for educational purposes and is not medical advice. If you take prescription medications, are pregnant or nursing, have a chronic health condition, or are scheduled for surgery, talk to your doctor or pharmacist before starting any supplement on this list. That guidance shows up again at the end, but it matters most at the beginning.
The supplements on this list are sorted into three evidence tiers, and the tier each one lands in is based on a few specific things. Strong-evidence supplements have multiple human randomized controlled trials behind them, plus at least one credible meta-analysis or systematic review showing a reproducible effect on either a recognized inflammatory biomarker — like high-sensitivity C-reactive protein, interleukin-6, or tumor necrosis factor-alpha — or a clinical endpoint such as joint pain, WOMAC scores in osteoarthritis, or disease activity in rheumatoid arthritis.
Moderate-evidence supplements have at least one credible meta-analysis showing benefit, but with real caveats. The trials might be smaller, the populations narrower, or the results mixed across different markers. Limited or emerging-evidence supplements show some positive signals in human trials, but the effect is small, restricted to a specific context, or based on a handful of studies that used different methods.
A few editorial choices to be transparent about: animal-only data and cell-culture results are not enough to earn a place on this list, no matter how exciting the mechanism looks. Trials funded entirely by a supplement manufacturer without independent replication are also discounted. And the dose information you will see throughout reflects the dose used in the clinical trials, not the suggested serving size on a typical supplement label, which often falls below the studied range.
If you only have a minute, this is the table to bookmark. The full discussion of each supplement is below, but here is the at-a-glance version you can use to baseline your expectations and decide what to read in more depth.
Omega-3 fatty acids — specifically the marine forms EPA (eicosapentaenoic acid) and DHA (docosahexaenoic acid) — have by far the largest body of human randomized trial evidence of any anti-inflammatory supplement on this list. They are not a fringe option or a wellness fad. They are one of the most heavily studied compounds in nutritional medicine, and the evidence for their effect on inflammation is unusually consistent in the populations most likely to need it.
The strongest signal shows up in people who already have inflammation. A systematic review and meta-analysis of omega-3 supplementation in diabetic and cardiovascular disease patients found improvements in several inflammatory biomarkers and lipid parameters compared with controls. Separate meta-analyses in cancer patients and in patients hospitalized with COVID-19 reported significant reductions in C-reactive protein versus placebo or usual care. In healthy people with normal baseline inflammation, the picture is more muted — the effect tends to be smaller and less consistent. This is an important nuance that often gets lost. Omega-3 works best when inflammation is already elevated, which is exactly when most people are looking for help.
The mechanism is also unusually well understood. EPA and DHA are the chemical precursors to a family of molecules called specialized pro-resolving mediators, including resolvins, protectins, and maresins. These compounds actively resolve inflammation rather than simply suppressing it, which sets omega-3 apart from drugs like NSAIDs that work by blocking inflammatory pathways. The clinical doses that show benefit in trials typically fall between one and three grams per day of combined EPA and DHA, and that range matters because most over-the-counter fish oil softgels deliver substantially less than one gram per capsule once you factor out the non-EPA, non-DHA fish oil content.
Two safety findings deserve attention. First, at very high doses — around 4 grams per day — omega-3 supplementation has been associated with an increased risk of atrial fibrillation in cardiovascular outcome trials. Second, omega-3 mildly thins the blood, which matters if you take warfarin, a DOAC like apixaban or rivaroxaban, an antiplatelet drug like clopidogrel, or even daily aspirin. Both issues are dose-related and most relevant at the higher doses used in cardiovascular trials.
If you are choosing a product, look for triglyceride or re-esterified triglyceride forms (better absorbed than ethyl esters) and third-party certification such as IFOS, USP, or NSF, which reduces the risk of oxidation and heavy-metal contamination. Omega-3 is most worth considering if your hs-CRP is elevated, if you have cardiovascular risk factors, if you are managing rheumatoid arthritis under a clinician's care, or if you have ongoing joint or muscle inflammation that is not responding to lifestyle changes alone.
Curcumin is the principal active compound in turmeric, the bright orange spice you probably already have in your kitchen. It is also the most studied plant-derived anti-inflammatory in modern clinical research, with a steady accumulation of randomized trials and meta-analyses across the past decade. The evidence is dense enough now that curcumin has earned its place alongside omega-3 in the strong-evidence tier, though there are some practical considerations that often get missed.
In knee osteoarthritis, a recent systematic review and meta-analysis of randomized controlled trials found that curcumin supplementation significantly reduced serum C-reactive protein and tumor necrosis factor-alpha compared with placebo. Similar findings have emerged from meta-analyses in rheumatoid arthritis (improvements in disease activity and inflammatory markers), inflammatory bowel disease (curcumin as an adjunctive therapy alongside standard care), and across diverse populations in broader umbrella reviews. The mechanism is multi-pathway: curcumin modulates NF-κB signaling, inhibits COX-2 expression, and dampens pro-inflammatory cytokine production, with additional antioxidant effects layered on top.
Here is where it gets practical, and where most listicles let readers down. Standard curcumin powder is notoriously poorly absorbed when taken orally. The amount that actually ends up in your bloodstream from a basic turmeric capsule is so low that some pharmacology studies struggle to measure it. This is why a 500 mg dose of plain curcumin and a 500 mg dose of an enhanced formulation are not the same thing in any meaningful sense. Several manufacturing approaches solve this problem differently. Combining curcumin with piperine (black pepper extract) increases its bioavailability roughly twentyfold. Phospholipid phytosomes such as Meriva and essential-oil-enriched curcumin like BCM-95 (also called Curcugreen) each improve absorption substantially. Theracurmin, a submicron colloidal dispersion, achieves the highest peak plasma curcumin levels in head-to-head pharmacokinetic comparisons. Longvida uses a solid lipid particle designed for both systemic and brain bioavailability. If you are going to take curcumin, choose a formulation with documented absorption data and skip the generic turmeric powder capsules.
There is also a newer safety story you should know about. Case reports and a published case series from the Drug-Induced Liver Injury Network — along with entries in the NIH LiverTox database — describe clinically apparent liver injury associated with turmeric and curcumin supplements, often involving the high-bioavailability or piperine-enhanced products that have become popular in recent years. The mechanism is not fully understood, but enhanced absorption may also mean enhanced hepatic exposure. Traditional turmeric used in cooking has a long history of safe use; the concern is concentrated supplements. If you have any history of liver disease, hepatitis, or take medications that are hepatotoxic, talk to your doctor before adding a curcumin supplement and let them know if you experience fatigue, jaundice, dark urine, or right-upper-quadrant abdominal pain after starting one.
Beyond the liver consideration, curcumin can mildly increase bleeding risk, may cause GI upset at higher doses, and can affect the metabolism of drugs cleared through the CYP3A4 enzyme system — relevant for tacrolimus, certain statins, and several chemotherapy agents. Curcumin is most worth considering if you are managing osteoarthritis, looking for an adjunct in rheumatoid arthritis or inflammatory bowel disease under specialist care, or addressing metabolic inflammation alongside lifestyle changes.
Ginger does not get the same press as turmeric, but its anti-inflammatory evidence is solid enough to land it firmly in the moderate-evidence tier, particularly for osteoarthritis. The most cited meta-analysis on this topic — a pooled analysis of five randomized placebo-controlled trials in osteoarthritis patients covering roughly 593 patients — found that oral ginger significantly reduced pain and improved physical function compared with placebo, with a standardized mean difference in pain of about negative 0.30. That is a modest effect, but it is real and replicable. A more recent GRADE-assessed systematic review and dose-response meta-analysis of ginger supplementation in adults reported antioxidant and anti-inflammatory effects across a range of doses, with biomarker improvements scaling with the amount taken.
The mechanism is overlapping with how some prescription anti-inflammatories work. Ginger inhibits lipoxygenase, which suppresses pro-inflammatory leukotriene synthesis. It reduces TNF-α expression in activated synoviocytes — the cells that line your joints and drive inflammatory pain. And it modulates cyclooxygenase activity, the same pathway NSAIDs like ibuprofen act on. That overlap is part of why ginger shows up in joint research so consistently.
Most clinical trials have used between one and two grams per day of standardized ginger extract, and up to two grams daily is generally considered safe. Side effects tend to be mild GI symptoms — occasional heartburn or stomach upset. At doses above two grams per day, ginger can have a measurable blood-thinning effect, which is worth knowing if you are on an anticoagulant or antiplatelet medication. Ginger is most worth considering if you are dealing with osteoarthritis pain, exercise-related joint inflammation, or low-level systemic inflammation that has not responded well to dietary changes alone.
Boswellia serrata, an extract from a tree native to India and parts of the Middle East, is one of the most underrated compounds in the joint-health space. Most people have never heard of it, but the clinical evidence for osteoarthritis is meaningful. A systematic review and meta-analysis of seven clinical trials covering 545 patients found significant improvements over placebo or active control in visual analog scale pain scores, WOMAC pain scores, and WOMAC stiffness scores. The effect sizes were not trivial — pain reductions averaged around 14 points on the WOMAC scale, and stiffness improvements were similar.
The active ingredients are a family of molecules called boswellic acids, and the most pharmacologically active is AKBA, short for 3-O-acetyl-11-keto-beta-boswellic acid. AKBA is a potent inhibitor of 5-lipoxygenase, an enzyme central to producing leukotrienes that drive inflammation in joints and elsewhere. This mechanism is distinct from how omega-3, curcumin, or NSAIDs work, which is part of why boswellia is sometimes used in combination protocols.
If you are choosing a boswellia product, total milligrams matter less than the standardization. Look for an extract standardized to 10–30% AKBA, or one of the branded enriched formulations like 5-Loxin or Aflapin that have been used in published trials. Most effective doses fall between 100 and 500 mg per day of standardized extract. Boswellia is generally well tolerated, with mild GI upset being the most common side effect; studies have not shown toxic effects at higher doses, but it may interact with immunomodulatory medications, which matters if you have an autoimmune diagnosis. Consider boswellia if knee osteoarthritis or other inflammatory joint conditions are your primary concern.
Vitamin D is the supplement that most needs an honest framing on this list, because the popular story — "everyone should take vitamin D for inflammation" — is not quite what the research supports. The defensible version is more specific: vitamin D supplementation reduces inflammation primarily in people who are deficient, and supplementing a vitamin-D-replete person is unlikely to produce a meaningful anti-inflammatory effect.
A meta-analysis of randomized controlled trials examining vitamin D supplementation and high-sensitivity C-reactive protein concluded that vitamin D modestly reduced circulating hs-CRP overall, with larger reductions in participants who had higher baseline inflammation. A separate bidirectional Mendelian randomization analysis found evidence consistent with low vitamin D status being causally associated with higher CRP, which supports the idea that correcting deficiency is the intervention that actually moves inflammation. At the same time, not every meta-analysis shows clean benefits across every biomarker — some have reported that vitamin D had no significant overall effect on CRP, IL-10, or TNF-α and actually raised IL-6, which underscores how heterogeneous the response is depending on who is studied and what is measured.
The practical implication is straightforward. If you have not had your 25-hydroxyvitamin D level checked recently, that is the place to start. Most clinical trials have used between 1,000 and 4,000 IU per day, and the Institute of Medicine's Tolerable Upper Intake Level for adults is 4,000 IU per day for routine supplementation. Higher correction-of-deficiency protocols (sometimes 50,000 IU once weekly under medical supervision) are sometimes used short-term to restore levels. Safety concerns are uncommon at standard doses but include hypercalcemia at sustained high intake without monitoring. Vitamin D is most worth considering if your 25(OH)D is below about 30 ng/mL, particularly if you also have an inflammatory or autoimmune condition.
Quercetin is a flavonoid found naturally in onions, apples, capers, and several other plant foods, and it has been studied as a supplement for inflammation, allergies, and exercise recovery. The evidence is mixed in a way that is honest to acknowledge: the picture trends positive at higher doses and in people who already have elevated inflammation, but the effects in mixed or healthy populations have been less consistent.
The most useful synthesis comes from a meta-analysis of randomized controlled trials examining quercetin and CRP, which reported a significant overall reduction in circulating CRP — a weighted mean difference around negative 0.33 mg/L — with the largest reductions in trials that used at least 500 mg per day. Earlier meta-analyses had found no significant overall effect on CRP, IL-6, or TNF-α in mixed populations, but subgroup analyses revealed CRP reductions specifically in participants with diagnosed disease. The pattern that emerges is that quercetin appears to work best when inflammation is already elevated and when the dose is high enough — generally between 500 and 1,000 mg per day.
Mechanistically, quercetin inhibits NF-κB signaling, modulates mast cell activity (which is part of why it is sometimes used in allergy-related inflammation), and acts as an antioxidant. Most people tolerate it well at typical doses. Possible interactions with quinolone antibiotics, cyclosporine, and some chemotherapy agents are worth flagging, and longer-term safety at doses above one gram per day is less well characterized in the literature. Consider quercetin if you are dealing with elevated baseline inflammation, allergy-driven symptoms, or want to add something modestly evidence-based to a stack that already includes the stronger options.
Resveratrol is the polyphenol that made wine famous, though almost all the clinical trial work uses concentrated supplements rather than red wine itself. The evidence base is moderate in size, smaller in magnitude than omega-3 or curcumin, but real — particularly in people with cardiovascular or metabolic disease.
A systematic review and meta-analysis of randomized controlled trials of resveratrol supplementation and inflammatory markers concluded that resveratrol significantly reduced CRP and TNF-α in adults, with effect sizes in the small-to-moderate range. The clearest benefits have appeared in patients with cardiovascular or metabolic conditions; effects in healthy populations have been smaller. Mechanistically, resveratrol activates SIRT1, modulates NF-κB, and acts as an antioxidant — pathways that overlap with several of the supplements above but produce less dramatic results in head-to-head terms.
Most clinical trials use doses between 150 and 500 mg per day. Bioavailability is low and variable, which is part of why some formulations use micronized or sustained-release delivery to improve absorption. Resveratrol is generally well tolerated, but it can cause GI symptoms at doses above one gram per day, can affect drugs metabolized through CYP3A4, and has some estrogenic activity at higher doses that matters for hormone-sensitive conditions. Consider resveratrol if you are addressing cardiovascular or metabolic inflammation, particularly as part of a broader strategy that includes diet and exercise.
A handful of supplements show up on almost every other "best anti-inflammatory" list, and it is worth saying briefly why they are not on this one. This is not a dismissal of all use cases — several of these have evidence for specific situations — but the criterion here is reproducible, systemic anti-inflammatory effect in human randomized trials.
Glucosamine and chondroitin have real evidence in moderate-to-severe knee osteoarthritis, particularly when used together (the GAIT trial showed a benefit in the moderate-to-severe pain subgroup). But these are joint-specific structural supplements rather than systemic anti-inflammatories, and they do not consistently move systemic biomarkers like hs-CRP. If your concern is specifically osteoarthritis, they may belong in a different conversation. If your concern is broader inflammation, they probably do not.
High-dose vitamin C is sometimes pitched as an anti-inflammatory, but the randomized trial evidence for chronic inflammation simply is not there. Correcting a documented deficiency is a separate matter. Spirulina has positive signals in some small trials but the body of evidence is still mostly preclinical or in heterogeneous designs that make pooled analysis difficult. Tart cherry has interesting data in exercise recovery and for urate-lowering effects relevant to gout, but its evidence base for systemic chronic inflammation is narrower than what would be needed for the rankings above. Bromelain, the enzyme complex from pineapple, has its strongest evidence in post-surgical inflammation (especially after dental procedures) rather than as a daily systemic anti-inflammatory; many of its broader inflammation trials are small or of low to moderate quality. Garlic, green tea extract, and SAMe each have supportive but narrower evidence that did not rise to the tiers above.
Then there are the multi-ingredient proprietary blends marketed as "anti-inflammatory complexes." These are nearly impossible to evaluate against any specific trial protocol because the individual doses are usually hidden inside a proprietary mix. If you cannot tell what dose of a specific compound you are actually taking, you cannot match it to the doses that have been studied, which means you cannot reasonably predict whether you will see any benefit.
Pick one supplement, give it eight to twelve weeks, and notice what changes. Most clinical trials measuring CRP reductions ran for roughly this duration, so anything shorter is premature. Faster judgment leads to bouncing from product to product without ever giving anything a fair test.
Match the supplement to the pattern. If joint pain is your main concern, curcumin or boswellia (or both) make sense as a starting point. If your hs-CRP is elevated and you have cardiovascular risk factors, omega-3 is the most defensible first move. If you are using inflammation as a recovery lens after intense training, the calculus is different. If you suspect deficiency-driven inflammation, vitamin D after testing is the right entry. For mixed metabolic inflammation, omega-3 first, then add curcumin if you want a second compound.
Get a baseline measurement if you can. A high-sensitivity CRP blood test is inexpensive in most U.S. labs (often under $30) and is widely covered by insurance with a clinician order. Retesting after twelve weeks tells you whether anything is actually moving. Joint Centers for Disease Control and American Heart Association guidance has long described how to interpret hs-CRP — less than 1 mg/L is considered low cardiovascular risk, 1 to 3 mg/L is average or intermediate risk, and above 3 mg/L is high risk for the kind of low-grade inflammation associated with chronic disease.
Diet, sleep, exercise, smoking status, and weight move inflammation more than any of the supplements on this list. That is not a hedge or a humility line — it is what the comparative evidence shows. Supplements are an adjunct to a baseline of healthy habits, not a substitute for them. And remember the formulation point from the curcumin section: a 500 mg plain curcumin capsule and 500 mg of Theracurmin, Meriva, BCM-95, or Longvida are not the same dose biologically. Read labels carefully and skip the multi-ingredient "anti-inflammatory blends" that hide individual doses behind proprietary mixes.
The supplements on this list are largely well tolerated by most people, but there are situations where extra care matters.
If you take an anticoagulant or antiplatelet drug — warfarin, apixaban, rivaroxaban, clopidogrel, or even daily aspirin — talk to your prescriber before adding omega-3, curcumin, ginger, or quercetin. All of these can modestly increase bleeding risk, and the effect compounds with prescription blood thinners.
If you have surgery scheduled, many surgeons recommend stopping fish oil, curcumin, and ginger seven to fourteen days before the procedure. Follow the specific guidance from your surgical team. If you have cardiovascular disease and are considering high-dose omega-3 (around 4 grams per day), discuss the atrial fibrillation signal with your cardiologist — the risk appears to be dose-related and most relevant at the higher therapeutic doses.
If you have a history of liver disease or hepatitis, take medications that are hepatotoxic, or notice symptoms like fatigue, jaundice, or right-upper-quadrant abdominal pain after starting a curcumin supplement, stop the product and check in with your physician. The DILIN turmeric cases have made this a real clinical consideration, especially with high-bioavailability or piperine-enhanced formulations.
If you have an autoimmune condition, immune-modulating supplements can interact unpredictably with immunosuppressants and biologic medications. Your rheumatologist or specialist should know about anything you are adding. If you are pregnant or nursing, most of the herbal supplements on this list have not been adequately studied in pregnancy; omega-3 is generally considered safe and is sometimes recommended in prenatal care, but curcumin, boswellia, and ginger in supplement (not culinary) doses should be cleared by an obstetric provider first. Bromelain is generally contraindicated in pregnancy. Children under twelve are not represented in most of these trials, and any supplementation in kids should go through a pediatric provider.
Finally, supplement quality varies more than most people realize. The FDA does not pre-approve dietary supplements for safety or efficacy. Independent third-party testing — USP, NSF, or IFOS certifications for fish oil — is the most practical safeguard you have against adulteration, mislabeling, and contamination.
About the Author
Angela Nightingale is a Senior Editor at Daily Vitality with over two decades of experience in digital publishing and health and wellness content. She specializes in turning complex, often-confusing health topics into clear, calm, and practical guidance that respects the reader's intelligence. Her work focuses on helping people feel informed and confident — never overwhelmed or alarmed — as they make everyday decisions about how they eat, move, rest, and age.
Omega-3 fatty acids (EPA + DHA) have the largest body of randomized trial evidence, particularly in people with elevated inflammation, cardiovascular disease, or autoimmune conditions. Curcumin is a close second and is the strongest plant-based option, with multiple recent meta-analyses showing biomarker reductions across diverse populations.
Most clinical trials measuring inflammatory biomarkers ran for eight to twelve weeks. Plan to give a supplement at least that long before deciding whether it is helping you. Shorter trials often miss real but gradual effects, and shorter personal experiments often lead to switching products before any compound has a fair chance.
Generally yes, and the combination is common in clinical practice — they act on different pathways and the evidence does not suggest a meaningful interaction between them. The one caution is that both can mildly increase bleeding risk, so if you take a blood thinner or have surgery scheduled, talk to your prescriber first.
Curcumin is the main active anti-inflammatory compound in turmeric, but turmeric root is only about 2 to 5 percent curcumin by weight. A standardized curcumin extract delivers far more of the active compound than turmeric powder does, and the bioavailability-enhanced formulations (Meriva, BCM-95, Theracurmin, Longvida) further increase how much actually reaches your bloodstream.
Omega-3, curcumin, ginger, and quercetin can all modestly increase bleeding risk. If you take warfarin, a DOAC like apixaban or rivaroxaban, an antiplatelet drug like clopidogrel, or daily aspirin, talk to your prescriber before starting any of these. The combined effect is not usually dramatic, but it is real and worth managing proactively.
A high-sensitivity CRP blood test gives you a useful baseline, and retesting after eight to twelve weeks of supplementation tells you whether your inflammation is actually moving. It is the simplest objective measure widely available, and most U.S. labs offer it for under $30 with a clinician order.
High-sensitivity C-reactive protein is the most widely used clinical marker of low-grade systemic inflammation. Levels below 1 mg/L are considered low cardiovascular risk, 1 to 3 mg/L is average or intermediate risk, and above 3 mg/L is high risk. Acute infections and injuries can temporarily push CRP much higher, so the test is most useful when you are not actively sick.
Standard curcumin powder is poorly absorbed when taken orally — so poorly that some pharmacokinetic studies struggle to measure it in serum at all. Bioavailability-enhanced formulations (curcumin with piperine, phospholipid phytosomes like Meriva, essential-oil-enriched curcumin like BCM-95, colloidal forms like Theracurmin, or solid lipid particles like Longvida) deliver multiples more curcumin into circulation, which is the dose that matters biologically.
Vitamin D supplementation appears to reduce inflammation primarily in people who are deficient. If your 25-hydroxyvitamin D level is normal, supplementing further is unlikely to move inflammation in any meaningful way. Get tested first, and only then decide whether supplementation makes sense for you.
Diet, sleep, regular movement, and stress management move inflammation more than any of the supplements on this list. A whole-food eating pattern rich in fatty fish, leafy greens, olive oil, berries, nuts, and legumes — and lower in ultra-processed foods, added sugars, and refined carbohydrates — is the foundation. Supplements work best as targeted additions, not replacements, for a diet built around these basics.
Stacking multiple supplements is common, but it is rarely necessary or wise to start everything at once. Adding one supplement at a time (and giving it eight to twelve weeks) makes it possible to actually tell what is doing what. If you are managing complex medication regimens or have multiple chronic conditions, a pharmacist or a clinician familiar with supplement interactions is worth consulting before building a stack.
EPA and DHA are both long-chain omega-3 fatty acids found in fatty fish and fish oil. EPA tends to be more directly involved in the anti-inflammatory effects measured in cardiovascular and joint trials, while DHA is particularly important for brain and eye tissue. Most fish oil products contain both; the specific EPA-to-DHA ratio matters less than the total combined dose for general anti-inflammatory purposes.
This article is for educational purposes and does not replace medical advice. If you take prescription medications, are pregnant or nursing, have a chronic health condition, or are scheduled for surgery, talk to your doctor or pharmacist before starting any supplement discussed here.
